Fig. 1

Postprandial dysmetabolism and its relation to atherosclerosis, diabetes, and associated vascular complications. Consuming high-caloric meals rich in fat and/or carbohydrates induces elevations in postprandial hyperglycemia and lipidemia. These metabolic disruptions trigger a cascade of oxidative stress and initiate an inflammatory response, potentially damaging endothelial cells. This association raises concerns about their contribution to the development of cardiovascular diseases and diabetic complications. ROS: Reactive oxygen species, TLR: Toll-like receptors, NF-κB: Nuclear factor-κB